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dc.contributor.authorOlewe , K Perez , Shehu Shagari Awandu, Munde,O Elly , Anyona,B Samuel, Raballah Evans, Asito S Amolo, Ogola Sidney, Ndenga Erick, Onyango, O Clinton , Rosemary Rochford, Douglas J Perkins, Ouma Collins
dc.date.accessioned2023-06-26T12:00:31Z
dc.date.available2023-06-26T12:00:31Z
dc.date.issued2023
dc.identifier.urihttps://repository.maseno.ac.ke/handle/123456789/5742
dc.descriptionhttps://bmccancer.biomedcentral.com/articles/10.1186/s12885-023-11063-2en_US
dc.description.abstractEpstein Barr virus (EBV)-associated endemic Burkitt’s Lymphoma pediatric cancer is associated with morbidity and mortality among children resident in holoendemic Plasmodium falciparum regions in western Kenya. P. falciparum exerts strong selection pressure on sickle cell trait (SCT), alpha thalassemia (-α3.7/αα), glucose-6-phosphate dehydrogenase (G6PD), and merozoite surface protein 2 (MSP-2) variants (FC27, 3D7) that confer reduced malarial disease severity. The current study tested the hypothesis that SCT, (-α3.7/αα), G6PD mutation and (MSP-2) variants (FC27, 3D7) are associated with an early age of EBV acquisition.en_US
dc.publisherBioMed Centralen_US
dc.subjectAlpha thalassemia, SCT, G6PD mutations, MSP-2, EBVen_US
dc.titleHemoglobinopathies, merozoite surface protein-2 gene polymorphisms, and acquisition of Epstein Barr virus among infants in Western Kenyaen_US
dc.typeArticleen_US


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